Search results for "GABAB receptor"

showing 10 items of 17 documents

Dysregulated Prefrontal Cortex Inhibition in Prepubescent and Adolescent Fragile X Mouse Model

2020

Changes in excitation and inhibition are associated with the pathobiology of neurodevelopmental disorders of intellectual disability and autism and are widely described in Fragile X syndrome (FXS). In the prefrontal cortex (PFC), essential for cognitive processing, excitatory connectivity and plasticity are found altered in the FXS mouse model, however, little is known about the state of inhibition. To that end, we investigated GABAergic signaling in the Fragile X Mental Retardation 1 (FMR1) knock out (Fmr1-KO) mouse medial PFC (mPFC). We report changes at the molecular, and functional levels of inhibition at three (prepubescence) and six (adolescence) postnatal weeks. Functional changes we…

0301 basic medicinecongenital hereditary and neonatal diseases and abnormalitiesGABAB receptorBiologyInhibitory postsynaptic potentiallcsh:RC321-57103 medical and health sciencesCellular and Molecular NeuroscienceGABA0302 clinical medicineNeurodevelopmental disorderSDG 3 - Good Health and Well-beingmedicinePrefrontal cortexMolecular Biologylcsh:Neurosciences. Biological psychiatry. NeuropsychiatryOriginal Researchprefrontal cortexGABAA receptormedicine.diseaseelectrophysiologyFMR1Fragile X syndrome030104 developmental biologyplasticityFragile XGABAergic/dk/atira/pure/sustainabledevelopmentgoals/good_health_and_well_beingNeuroscience030217 neurology & neurosurgeryNeuroscienceFrontiers in Molecular Neuroscience
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Biphasic effects of cannabinoids in anxiety responses: CB1 and GABA(B) receptors in the balance of GABAergic and glutamatergic neurotransmission

2012

Biphasic effects of cannabinoids have been shown in processes such as feeding behavior, motor activity, motivational processes and anxiety responses. Using two different tests for the characterization of anxiety-related behavior (elevated plus-maze and holeboard), we first identified in wild-type C57BL/6N mice, two doses of the synthetic CB1 cannabinoid receptor agonist CP-55,940 with anxiolytic (1 mug/kg) and anxiogenic properties (50 mug/kg), respectively. To clarify the role of CB1 receptors in this biphasic effect, both doses were applied to two different conditional CB1 receptor knockout (KO) mouse lines, GABA-CB1-KO (CB1 receptor inactivation in forebrain GABAergic neurons) and Glu-CB…

AgonistMaleCannabinoid receptormedicine.drug_classmedicine.medical_treatmentGlutamic AcidCyclopentanesPharmacologyGABAB receptorBiologyAnxietyMotor ActivityAnxiolyticSynaptic TransmissionGlutamatergicMiceReceptor Cannabinoid CB1medicineAnimalsGABA Agonistsgamma-Aminobutyric AcidPharmacologyMice KnockoutBehavior AnimalDose-Response Relationship DrugCannabinoidsfood and beveragesCyclohexanolsMice Inbred C57BLPsychiatry and Mental healthPyrimidinesAnxiogenicnervous systemReceptors GABA-BGABAergiclipids (amino acids peptides and proteins)Original ArticleCannabinoidpsychological phenomena and processes
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Control of cortical neuronal migration by glutamate and GABA

2015

Neuronal migration in the cortex is controlled by the paracrine action of the classical neurotransmitters glutamate and GABA. Glutamate controls radial migration of pyramidal neurons by acting primarily on NMDA receptors and regulates tangential migration of inhibitory interneurons by activating non-NMDA and NMDA receptors. GABA, acting on ionotropic GABAA-rho and GABAA receptors, has a dichotomic action on radially migrating neurons by acting as a GO signal in lower layers and as a STOP signal in upper cortical plate (CP), respectively. Metabotropic GABAB receptors promote radial migration into the CP and tangential migration of interneurons. Besides GABA, the endogenous GABAergic agonist …

Cerebral Cortexneuronal migrationNeuronal Migration DisordersGABAA receptorGlutamate receptorKainate receptorReview ArticleGABAB receptorBiologylcsh:RC321-571Cellular and Molecular NeuroscienceGABAMetabotropic receptornervous systemNMDA receptorGlutamateLong-term depressionNeurosciencelcsh:Neurosciences. Biological psychiatry. NeuropsychiatryIonotropic effectNeuroscienceFrontiers in Cellular Neuroscience
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Haploinsufficiency of Tsc2 Leads to Hyperexcitability of Medial Prefrontal Cortex via Weakening of Tonic GABAB Receptor-mediated Inhibition.

2020

Abstract Loss-of-function mutation in one of the tumor suppressor genes TSC1 or TSC2 is associated with several neurological and psychiatric diseases, including autism spectrum disorders (ASDs). As an imbalance between excitatory and inhibitory neurotransmission, E/I ratio is believed to contribute to the development of these disorders, we investigated synaptic transmission during the first postnatal month using the Tsc2+/− mouse model. Electrophysiological recordings were performed in acute brain slices of medial prefrontal cortex. E/I ratio at postnatal day (P) 15–19 is increased in Tsc2+/− mice as compared with wildtype (WT). At P25–30, facilitated GABAergic transmission reduces E/I rati…

Cognitive NeurosciencePrefrontal CortexMice TransgenicHaploinsufficiencyGABAB receptorNeurotransmissionInhibitory postsynaptic potentialSynaptic TransmissionTonic (physiology)03 medical and health sciencesCellular and Molecular Neurosciencechemistry.chemical_compound0302 clinical medicineTuberous Sclerosis Complex 2 ProteinAnimalsPrefrontal cortex030304 developmental biologyNeurons0303 health sciencesChemistryElectrophysiologyBaclofenReceptors GABA-BExcitatory postsynaptic potentialNeuroscience030217 neurology & neurosurgeryCerebral cortex (New York, N.Y. : 1991)
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Transporter-mediated replacement of extracellular glutamate for GABA in the developing murine neocortex

2013

During early development, cortical neurons migrate from their places of origin to their final destinations where they differentiate and establish synaptic connections. During corticogenesis, radially migrating cells move from deeper zone to the marginal zone, but they do not invade the latter. This "stop" function of the marginal zone is mediated by a number of factors, including glutamate and γ-aminobutyric acid (GABA), two main neurotransmitters in the central nervous system. In the marginal zone, GABA has been shown to be released via GABA transporters (GAT)-2/3, whereas glutamate transporters (EAATs) operate in the uptake mode. In this study, GABAergic postsynaptic currents (GPSCs) were…

GABA Plasma Membrane Transport ProteinsAmino Acid Transport System X-AGGlutamic AcidNeocortexBiologyGABAB receptorMicemedicineAnimalsGABA transporterGABAergic Neuronsgamma-Aminobutyric AcidNeocortexGeneral NeuroscienceSodiumGlutamate receptorDepolarizationSynaptic PotentialsMarginal zoneCell biologyMice Inbred C57BLmedicine.anatomical_structurebiology.proteinGABAergicGABA Uptake InhibitorsNeuroscienceIntracellularEuropean Journal of Neuroscience
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GABA transporters control GABAergic neurotransmission in the mouse subplate.

2015

The subplate is a transient layer between the cortical plate and intermediate zone in the developing cortex. Thalamo-cortical axons form temporary synapses on subplate neurons (SPns) before invading the cortical plate. Neuronal activity within the subplate is of critical importance for the development of neocortical circuits and architecture. Although both glutamatergic and GABAergic inputs on SPns were reported, short-term plasticity of GABAergic transmission has not been investigated yet. GABAergic postsynaptic currents (GPSCs) were recorded from SPns in coronal neocortical slices prepared from postnatal day 3-4 mice using whole-cell patch-clamp technique. Evoked GPSCs (eGPSCs) elicited b…

GABA Plasma Membrane Transport ProteinsGABA Plasma Membrane Transport ProteinsPatch-Clamp TechniquesGABAB receptorBiologyNeurotransmissionSynaptic Transmissiongamma-Aminobutyric acidTissue Culture TechniquesGlutamatergicSubplatemedicinePremovement neuronal activityAnimalsgamma-Aminobutyric AcidGeneral NeuroscienceSomatosensory CortexSynaptic PotentialsReceptors GABA-AElectric StimulationMice Inbred C57BLmedicine.anatomical_structureReceptors GABA-BGABAergicNeurosciencemedicine.drugCentral Nervous System AgentsNeuroscience
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Age-related differences of γ-aminobutyric acid (GABA)ergic transmission in human colonic smooth muscle.

2021

Background: Enteric neurons undergo to functional changes during aging. We investigated the possible age-associated differences in enteric γ-aminobutyric acid (GABA)ergic transmission evaluating function and distribution of GABAergic receptors in human colon. Methods: Mechanical responses to GABA and GABA receptor agonists on slow phasic contractions were examined in vitro as changes in isometric tension in colonic muscle strips from young (<65 years old) and aged patients (>65 years old). GABAergic receptor expression was assessed by quantitative RT-PCR. Key Results: In both preparations GABA induced an excitatory effect, consisting in an increase in the basal tone, antagonized by th…

GABAA receptor subunitmedicine.medical_specialtyAgingintestinal motilityPhysiologyColonReceptor expressionTetrodotoxinGABAB receptorSettore BIO/09 - Fisiologiachemistry.chemical_compoundGABAPhaclofenGABA receptorSettore BIO/13 - Biologia ApplicataInternal medicinemedicineHumansgamma-Aminobutyric AcidAgedEndocrine and Autonomic SystemsGABAA receptorGastroenterologyMuscle SmoothBicucullineReceptors GABA-AEndocrinologyGABAergic receptorsnervous systemMuscimolchemistryReceptors GABA-BGABAergicmedicine.drugMuscle ContractionNeurogastroenterology and motility : the official journal of the European Gastrointestinal Motility Society
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Intracellular Na+ concentration influences short-term plasticity of glutamate transporter-mediated currents in neocortical astrocytes.

2012

Fast synaptic transmission requires a rapid clearance of the released neurotransmitter from the extracellular space. Glial glutamate transporters (excitatory amino acid transporters, EAATs) strongly contribute to glutamate removal. In this work, we investigated the paired-pulse plasticity of synaptically activated, glutamate transporter-mediated currents (STCs) in cortical layer 2/3 astrocytes. STCs were elicited by local electrical stimulation in layer 4 in the presence of ionotropic glutamate (AMPA and NMDA), GABAA, and GABAB receptor antagonists. In experiments with low [Na+]i (5 mM) intrapipette solution, STCs elicited by paired-pulse stimulation demonstrated paired-pulse facilitation (…

Intracellular FluidPatch-Clamp TechniquesAmino Acid Transport System X-AGBiophysicsNipecotic AcidsAction PotentialsNeocortexAMPA receptorTetrodotoxinBiologyGABAB receptorAnisolesIn Vitro TechniquesSynaptic TransmissionGABA AntagonistsCellular and Molecular NeuroscienceMiceCadmium ChlorideEthers CyclicOximesmedicineGABA transporterAnimalsgamma-Aminobutyric AcidBenzofuransAspartic AcidNeuronal PlasticityGABAA receptorRhodaminesSodiumGlutamate receptorCalcium Channel BlockersElectric StimulationMice Inbred C57BLmedicine.anatomical_structureNeurologyAnimals NewbornAstrocytesExcitatory postsynaptic potentialBiophysicsbiology.proteinNMDA receptorNeuroscienceExcitatory Amino Acid AntagonistsAstrocyteSodium Channel BlockersGlia
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GABA receptors are involved in the modulation of the release of 5-hydroxytryptamine from the vascularly perfused small intestine of the guinea-pig

1989

Isolated small intestinal segments of the guinea-pig were perfused arterially and the release of 5-hydroxytryptamine (5-HT) and 5-hydroxyindoleacetic acid (5-HIAA) into the portal venous effluent was determined by HPLC with electrochemical detection. Test substances were applied intraarterially. Muscimol (1 microM) time dependently first increased then decreased the release of 5-HT and 5-HIAA. The stimulatory effect was prevented by tetrodotoxin (TTx) or scopolamine, indicating that it was mediated by the release of acetylcholine. Bicuculline concentration dependently decreased (1 microM) or increased (10, 50 microM) the release of 5-HT and 5-HIAA, indicating that endogenous GABA also activ…

MaleBaclofenSerotoninmedicine.medical_specialtyGuinea PigsTetrodotoxinIn Vitro TechniquesBiologyGABAB receptorBicucullineInhibitory postsynaptic potential5-Hydroxytryptophanchemistry.chemical_compoundInternal medicineIntestine SmallmedicineAnimalsReceptorPharmacologyMuscimolGABAA receptorOxotremorineMuscle SmoothHydroxyindoleacetic AcidBicucullineReceptors GABA-APerfusionEndocrinologynervous systemMuscimolchemistryFemaleSerotoninAcetylcholinemedicine.drugEuropean Journal of Pharmacology
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Comparative pharmacological activity of optical isomers of phenibut

2007

Phenibut (3-phenyl-4-aminobutyric acid) is a GABA (gamma-aminobutyric acid)-mimetic psychotropic drug which is clinically used in its racemic form. The aim of the present study was to compare the effects of racemic phenibut and its optical isomers in pharmacological tests and GABAB receptor binding studies. In pharmacological tests of locomotor activity, antidepressant and pain effects, S-phenibut was inactive in doses up to 500 mg/kg. In contrast, R-phenibut turned out to be two times more potent than racemic phenibut in most of the tests. In the forced swimming test, at a dose of 100 mg/kg only R-phenibut significantly decreased immobility time. Both R-phenibut and racemic phenibut showed…

MaleHot TemperaturePhenibutMotor ActivityPharmacologyGABAB receptorConflict PsychologicalGABA AntagonistsMicechemistry.chemical_compoundOrganophosphorus CompoundsReaction TimemedicineAnimalsMuscle StrengthGABA AgonistsPostural BalanceSwimminggamma-Aminobutyric AcidPain MeasurementPharmacologyAnalgesicsMice Inbred ICRPsychotropic DrugsDepressionAntagonistStereoisomerismBiological activityAntidepressive AgentsPsychotropic drugBaclofenReceptors GABA-BchemistryMice Inbred CBAEnantiomerPsychomotor Performancemedicine.drugBehavioural despair testEuropean Journal of Pharmacology
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